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Standard Care

Is a Lipid Panel Worth It?

Half of heart attacks occur in people with "normal" cholesterol. Standard lipid panels miss critical risk markers—ApoB counts the particles that actually matter, not just the cholesterol they carry.

Published 23 January 2026 · Updated 27 January 2026 · 10 min read

42%

of Australian adults have high cholesterol—yet standard testing misses 10-20% of people at elevated cardiovascular risk

Source: [1]

The Short Answer

Yes—but standard testing alone isn't enough. Lipid panels are foundational for cardiovascular risk assessment. The RACGP recommends testing every 5 years from age 45 (35 for Aboriginal and Torres Strait Islander peoples). But here's what matters: standard LDL cholesterol measures cargo, not vehicles. ApoB directly counts atherogenic particles—the actual drivers of plaque formation. For anyone serious about optimising cardiovascular health, ApoB isn't optional; it's the superior marker.[1][2]

The Australian Context

Cardiovascular disease kills one Australian every 12 minutes. With 42% of adults having high cholesterol and CVD claiming 1 in 4 Australian lives, lipid testing is fundamental to prevention.[1]

But here's the uncomfortable reality: roughly half of heart attacks occur in people with cholesterol in the "normal" range.[3] This isn't because cholesterol doesn't matter—it's because standard lipid testing measures the wrong thing.

The "Normal Cholesterol" Trap

Standard lipid panels measure cholesterol content. They don't count particles—and it's particles that penetrate artery walls and form plaque.

Two people with identical LDL-C can have wildly different particle counts. Person A might have 1,000 large particles. Person B might have 2,000 small particles. Same cholesterol, double the risk.

This is why ApoB—which directly counts atherogenic particles—outperforms LDL-C in 9 of 9 studies for predicting cardiovascular events.[2]

What's Measured in a Lipid Panel

A standard lipid panel measures several markers. Understanding what each represents—and what they miss—is critical.

Risk FactorWhy It Matters
The sum of all cholesterol in your blood. A blunt instrument—tells you almost nothing about actual risk. Australian guidelines have moved away from using it as a treatment target.
Low-density lipoprotein cholesterol. Called "bad" cholesterol because it's associated with plaque formation. But here's the catch: LDL-C measures cholesterol content, not particle number. It's usually calculated, not measured, using an equation that fails when triglycerides are elevated.[4]

The LDL Limitation Nobody Mentions

Here's what most people—and many GPs—don't realise: LDL cholesterol is rarely measured directly. It's calculated using the Friedewald equation from your total cholesterol, HDL, and triglycerides.

The equation assumes a fixed ratio that doesn't hold when triglycerides are elevated. And it measures cholesterol content, not the number of particles actually causing disease.

The Friedewald Problem

The Friedewald equation systematically underestimates LDL cholesterol when accuracy matters most:[4]

  • Triglycerides 1.7-2.3 mmol/L: LDL underestimated by ~0.23 mmol/L
  • Triglycerides 2.3-4.5 mmol/L: LDL underestimated by ~0.47 mmol/L
  • Triglycerides >4.5 mmol/L: Calculation considered unreliable

"The Friedewald equation tends to underestimate LDL-C most when accuracy is most crucial," notes Johns Hopkins researcher Dr Seth Martin. Patients with metabolic syndrome—those at highest risk—get the least accurate results.[4]

Why ApoB Is Superior

Apolipoprotein B (ApoB) is a protein found on every atherogenic particle in your blood—LDL, VLDL, IDL, and Lp(a). There is exactly one ApoB molecule per particle.[2]

This means measuring ApoB gives you a direct particle count. Not an estimate. Not a calculation that fails when triglycerides are high. A direct measurement of the vehicles actually delivering cholesterol to your artery walls.

LDL-C: Measures Cargo

  • Measures cholesterol content in LDL particles
  • Usually calculated, not measured directly
  • Inaccurate when triglycerides >1.7 mmol/L
  • Misses VLDL and IDL particle contribution
  • Standard of care for 40+ years

ApoB: Counts Vehicles

  • Directly counts all atherogenic particles
  • Directly measured—no calculation needed
  • Accurate regardless of triglyceride levels
  • Captures LDL + VLDL + IDL + Lp(a)
  • Superior predictor in 9 of 9 studies[2]

The Discordance Problem

In 10-20% of people, LDL-C and ApoB tell radically different stories. This is called discordance—and it's where standard lipid testing fails most catastrophically.[5]

When LDL-C Lies

The mass of cholesterol per particle varies substantially. In discordant cases:[5]

  • "Normal" LDL-C but elevated ApoB: Many small, cholesterol-depleted particles. Higher risk than LDL suggests. Common in metabolic syndrome.
  • Elevated LDL-C but normal ApoB: Fewer, larger particles carrying more cholesterol each. Lower risk than LDL suggests.

A 2025 UK Biobank study of 41,099 participants found that even at just 2% discordance, cardiovascular risk was elevated. At 30% discordance, hazard ratios reached 2.5x for coronary artery disease.[6]

When discordant, ApoB is always the better predictor. Not sometimes. Always.

What the Guidelines Say

International guidelines have shifted decisively toward ApoB. Australian guidelines are lagging—but the evidence is unambiguous.

European Atherosclerosis Society (2025)

  • "ApoB is recommended as the primary target for lipid-lowering therapy"
  • Particularly in patients with elevated triglycerides, diabetes, or metabolic syndrome
  • Recognises superior predictive accuracy vs LDL-C

National Lipid Association (2024)

  • "ApoB has been shown to be superior to LDL-C in risk assessment"
  • Recommends ApoB both before and during treatment
  • Proposes stratified targets: <90 mg/dL intermediate risk, <70 high risk, <60 very high risk

Australian Guidelines

  • RACGP and Heart Foundation guidelines still focus on LDL-C
  • Updated guidance expected as international evidence accumulates
  • Most Australian GPs unfamiliar with ApoB targets and management

Why Australian Guidelines Lag

Australian guideline updates follow a rigorous, slow process—rightly so for clinical care. But this means local recommendations can lag international evidence by years.

The European Atherosclerosis Society and National Lipid Association have reviewed the same evidence and reached clear conclusions: ApoB is superior. Australian guidelines will eventually follow—the question is whether you wait for them to catch up or act on the evidence now.[7][8]

When Triglycerides Make LDL Unreliable

If your triglycerides are elevated—and 30% of Australian adults have levels >1.7 mmol/L—your LDL-C result is unreliable. Not slightly off. Systematically underestimated.

Low Triglycerides (<1.2 mmol/L)

  • Friedewald equation reasonably accurate
  • LDL-C and ApoB usually concordant
  • Standard lipid panel likely sufficient
  • ApoB still superior but gap smaller

Elevated Triglycerides (>1.7 mmol/L)

  • Friedewald equation systematically underestimates LDL
  • LDL-C and ApoB frequently discordant
  • Standard lipid panel inadequate
  • ApoB testing essential for accurate risk assessment
If you have metabolic syndrome, insulin resistance, type 2 diabetes, or obesity—all conditions that elevate triglycerides—standard lipid testing is not sufficient. ApoB is the only marker that remains accurate.

Do You Need to Fast?

For most people, fasting is no longer required. Current Australian guidelines accept non-fasting lipid results for routine cardiovascular risk assessment.[1]

Non-Fasting Acceptable

  • Routine cardiovascular risk assessment
  • General lipid screening
  • Most healthy adults
  • When fasting would cause you to skip testing

Fasting Preferred

  • Triglycerides previously very high (>4.5 mmol/L)
  • Monitoring diabetes or metabolic syndrome
  • When GP specifically requests it
  • For most precise LDL calculation

Understanding Your Targets

Standard guidelines provide LDL-C targets. But if you're optimising based on ApoB—the superior marker—here are the evidence-based ranges:[7][8]

ApoB <60 mg/dL (0.6 g/L)Optimal for very high risk

Target for those with recent cardiovascular event, multiple risk factors, or established atherosclerosis. Associated with lowest event rates. Requires medication; lifestyle alone cannot achieve this.

ApoB 60-70 mg/dL (0.6-0.7 g/L)Optimal for high risk

Target for existing cardiovascular disease, diabetes with complications, or very high calculated risk. National Lipid Association recommendation for high-risk individuals.

ApoB 70-90 mg/dL (0.7-0.9 g/L)Target for moderate risk

Appropriate for moderate-risk individuals. Many preventive cardiologists aim for this range proactively rather than waiting for disease.

ApoB 90-100 mg/dL (0.9-1.0 g/L)Acceptable for low risk

Generally acceptable for those without cardiovascular risk factors. Still lower than population average but higher than optimal.

ApoB >100 mg/dL (1.0+ g/L)Elevated

Associated with increased cardiovascular risk. Discuss management options with your health practitioner, particularly if other risk factors present.

Australian GPs and ApoB Targets

Most Australian GPs are unfamiliar with ApoB targets because RACGP guidelines don't specify them. The ranges above are based on European Atherosclerosis Society and National Lipid Association consensus—guidelines that reflect current evidence.

If you test ApoB, be prepared to educate your GP or work with a preventive cardiologist who routinely uses particle-based targets. This isn't a criticism of GPs—it's a lag in local guideline updates.[9]

How Often to Test

Testing frequency depends on your baseline results, risk factors, and whether you're monitoring treatment.

Baseline Screening

  • Every 5 years from age 45[9]
  • From age 35 for Aboriginal and Torres Strait Islander peoples
  • Earlier if family history of premature heart disease
  • Include ApoB for accurate particle assessment

With Risk Factors

  • Every 1-2 years with high blood pressure or metabolic syndrome
  • Annually with diabetes or prediabetes
  • More frequently if borderline results
  • ApoB essential—standard panel insufficient

Monitoring Treatment

  • 6-8 weeks after starting lipid-lowering medication
  • Then every 6-12 months once stable
  • Sooner if side effects or dose changes
  • ApoB superior to LDL-C for verifying treatment efficacy

After Lifestyle Changes

  • Recheck 2-3 months after dietary changes
  • After significant weight loss (5-10%)
  • When starting regular exercise programme
  • ApoB shows true impact on particle number

Is It Worth the Cost?

Standard lipid panels are Medicare-covered. ApoB testing costs $30-50 out-of-pocket. Here's when the additional cost delivers genuine value:

✗ Probably Skip If...

  • You had a complete lipid panel with ApoB in the last 12 months with optimal results
  • You're under 35 with no risk factors and low triglycerides (<1.2 mmol/L)
  • Cost is a significant barrier and your risk factors are minimal

✓ Worth Considering If...

  • You're 45+ and haven't had recent testing (35+ for Aboriginal and Torres Strait Islander peoples)[9]
  • Metabolic syndrome, insulin resistance, or type 2 diabetes—LDL-C calculation unreliable[4]
  • Elevated triglycerides (>1.7 mmol/L) making standard testing inaccurate
  • Family history of early heart disease, particularly with 'normal' cholesterol
  • You want the most accurate cardiovascular risk assessment available[2]
  • You're on statin therapy and want to verify residual particle-level risk[7]
  • You're serious about cardiovascular optimisation—not just disease prevention

Our Recommendation: Always Include ApoB

At Clarity Labs, we include ApoB in our Heart Health panel because standard lipid testing is insufficient for accurate risk assessment. The evidence is unambiguous: ApoB outperforms LDL-C for predicting cardiovascular events.

If you're testing lipids, you deserve accurate information. ApoB provides that. LDL-C alone does not.

The Bottom Line

Lipid testing is essential—but standard testing alone is insufficient.

With 42% of Australian adults having high cholesterol and CVD claiming one life every 12 minutes, knowing your lipid levels is fundamental. But standard LDL cholesterol measures cargo, not vehicles. It's calculated using an equation that fails when triglycerides are elevated—precisely when accuracy matters most.

ApoB directly counts atherogenic particles. It outperforms LDL-C in 9 of 9 studies. When LDL-C and ApoB disagree—which happens in 10-20% of people—ApoB is always the better predictor. The European Atherosclerosis Society and National Lipid Association have reviewed the evidence and reached clear conclusions: ApoB is superior.

Who needs ApoB testing?

  • Anyone with metabolic syndrome, insulin resistance, or type 2 diabetes
  • Anyone with elevated triglycerides (>1.7 mmol/L)
  • Anyone with family history of early heart disease
  • Anyone serious about accurate cardiovascular risk assessment

Set realistic expectations: Most Australian GPs aren't familiar with ApoB targets because local guidelines haven't updated yet. You may need to work with a preventive cardiologist or advocate for yourself. Medicare doesn't cover it ($30-50 out-of-pocket).

But if you're testing lipids, you deserve accurate information. ApoB provides that. Standard testing alone does not.

Frequently Asked Questions

LDL-C measures cholesterol content in particles. ApoB directly counts the number of atherogenic particles in your blood. Since each particle has exactly one ApoB molecule, ApoB gives you a direct particle count. Research shows particle number predicts cardiovascular events better than cholesterol content—ApoB outperformed LDL-C in 9 of 9 studies. When LDL-C and ApoB disagree (10-20% of people), ApoB is always the better predictor.

Based on European Atherosclerosis Society and National Lipid Association guidelines: <90 mg/dL for intermediate-risk individuals, <70 mg/dL for high-risk individuals (existing cardiovascular disease, diabetes, or very high calculated risk), and <60 mg/dL for very high-risk patients with recent cardiovascular events or multiple risk factors. Australian guidelines don't yet specify ApoB targets—these are based on international evidence.

No, ApoB testing is not currently covered by Medicare in Australia. You'll typically pay $30-50 out-of-pocket. Standard lipid panels (total cholesterol, LDL-C, HDL, triglycerides) are Medicare-covered when ordered as part of cardiovascular risk assessment.

Three reasons: (1) RACGP guidelines still focus on LDL-C targets rather than ApoB, (2) it's not Medicare-covered so GPs are hesitant to request out-of-pocket tests, and (3) most GPs weren't trained on ApoB and may not know optimal targets or how to incorporate results into management. European and American guidelines have shifted to ApoB, but Australian guidelines lag by several years.

ApoB is essential when standard LDL-C is likely to be inaccurate or discordant: (1) Metabolic syndrome or insulin resistance—small, dense particles make discordance common, (2) Elevated triglycerides (>1.7 mmol/L)—Friedewald equation systematically underestimates LDL, (3) Type 2 diabetes—diabetic dyslipidaemia features particle abnormalities LDL-C misses, (4) Family history of early heart disease—may indicate inherited particle phenotype. In these situations, standard testing is insufficient.

Yes—this is called discordance, and it occurs in 10-20% of people. You might have many small particles each carrying less cholesterol, resulting in "normal" LDL-C but elevated ApoB. This indicates higher cardiovascular risk than your LDL suggests. A 2025 UK Biobank study found that at 30% discordance, cardiovascular risk increased by 2.5x. When discordant, ApoB is always the better predictor.

For most people, fasting is no longer required. Current Australian guidelines accept non-fasting lipid results for routine cardiovascular risk assessment. Fasting may still be preferred if your triglycerides were previously very high (>4.5 mmol/L), if you're monitoring diabetes or metabolic syndrome, or if your GP specifically requests it for the most precise LDL calculation.

RACGP recommends lipid testing every 5 years from age 45 (35 for Aboriginal and Torres Strait Islander peoples). If you have risk factors like diabetes, high blood pressure, or elevated triglycerides, test annually. If you're on lipid-lowering medication, recheck 6-8 weeks after starting, then every 6-12 months once stable. Include ApoB in all lipid testing for accurate particle assessment—particularly if you have metabolic syndrome or elevated triglycerides.

Three options: (1) Ask your GP to add ApoB to your lipid panel request—they can order it and you pay out-of-pocket (~$30-50), (2) See a preventive cardiologist who routinely includes ApoB in advanced lipid assessment, or (3) Order through a direct-to-consumer service like Clarity Labs. Any NATA-accredited pathology lab can run the test.

Disclaimer:This information is educational only and not medical advice. Results should be interpreted by your health practitioner in the context of your symptoms and health history. Treatment decisions should be made with your doctor or specialist.

  1. Heart Foundation. Guideline for the diagnosis and management of hypertension in adults and lipid management. 2024.
  2. Sniderman AD et al. Apolipoprotein B Particles and Cardiovascular Disease: A Narrative Review. JAMA Cardiology, 2019;4(12):1287-1295.
  3. Sachdeva A et al. Lipid levels in patients hospitalized with coronary artery disease: An analysis of 136,905 hospitalizations. American Heart Journal, 2009;157(1):111-117.
  4. Martin SS et al. Comparison of a Novel Method vs the Friedewald Equation for Estimating LDL Cholesterol. JAMA, 2013;310(19):2061-2068.
  5. Mora S et al. Discordance of Low-Density Lipoprotein Cholesterol and Particle Number. Journal of the American College of Cardiology, 2016;67(21):2454-2463.
  6. Kazi S et al. Apolipoprotein B outperforms LDL particle number as a marker of cardiovascular risk in the UK Biobank. PubMed, 2025.
  7. Mach F et al. 2025 Focused Update: ESC/EAS Guidelines for the management of dyslipidaemias. European Heart Journal, 2025.
  8. Wilson DP et al. Role of apolipoprotein B in the clinical management of cardiovascular risk: Expert Clinical Consensus from the National Lipid Association. Journal of Clinical Lipidology, 2024.
  9. RACGP. Guidelines for preventive activities in general practice (Red Book). 9th edition, updated December 2024. Cardiovascular disease risk assessment.
  10. Australian Institute of Health and Welfare. Heart, stroke and vascular disease: Australian facts. AIHW, 2024.